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The results revealed that the rhGPx3 mutant existed as a monomer, which was different from native human GPx3
Copyright: The Author(s) About this article Cite this article Conrad, R.J., Mondo, J.A., Wang, M.L
NFB-activated astroglial release of complement C3 compromises neuronal morphology and function associated with Alzheimer's disease
Y., Zhang, X
