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NNMT inhibition increases intracellular concentrations of NAD + and SAM in differentiated adipocytes Figure 3A outlines the major elements of the mammalian NAD+ salvage pathway using NA as the starting substrate.[27] Since the NNMT inhibitor 5-amino-1MQ significantly reduced intracellular 1-MNA concentrations (Figures 2B and 2C), we hypothesized that NNMT inhibition in adipocytes would increase intracellular concentrations of the co-substrates NA and SAM and shunt more NA into the NAD + salvage cycle

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Conclusions Although many points remain to be further shown or explained, particularly due to the use of the animal models and their general translational significance, this review attempts to resolve the shortage that the pleiotropic beneficial effects of BPC 157 may not fulfill the general standard neurotransmitter criteria, in classic terms

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