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Description
This is attributed by downregulation of CHAC1, an enzyme that specifically degrades intracellular GSH

Insulin resistance and cytokine production are key mechanisms leading to fatty change in the liver and may produce nonalcoholic steatohepatitis (NASH)
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In this study, we investigated the effects of exogenous GSH on the phenylpropanoid biosynthesis pathway and plant hormone signal transduction pathway in potatoes under Cd stress to explore new ideas for how potatoes respond to Cd stress