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In addition, the GSH redox status can affect the expression and activity of many factors important for cell cycle progression

Analysis of the lipid deposition patterns in immune cells revealed that, compared with myeloid cells, CD4+ T cells were more enriched in PUFA-PLs (substrates of LPO), making these cells highly sensitive to ferroptosis (56)

Fate mapping analysis reveals that adult microglia derive from primitive macrophages

Furthermore, glutathione modulates synaptic function by affecting redox-sensitive signalling pathways, which are crucial for maintaining neuronal excitability and synaptic plasticity [23]