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Impact: inflammation of the oviduct, cystic ovaries, and reduced synthesis of yolk precursors in the liver, resulting in a sharp drop in egg production and poor internal egg quality
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These findings corroborate with a previous functional study conducted by Sharma and colleagues [50], who demonstrated that PPAR silencing in endothelial cells leads to profound mitochondrial dysfunction by accentuating transmembrane depolarization and ROS production, lowering the cellular ATP levels and hampering the eNOS/Hsp90 axis, critical for energy metabolism regulation
