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This occurred along with the recovery of the NO-level to normal values through BPC 157 therapy (regularly, the NO-level decreased during ischemia and increased during reperfusion) and counteraction to normal values of the increased MDA values (increased in ischemia and even more in reperfusion) [194] (see Section 3.2)
Authors contributions A.B., A.N., N.D.D., and G.R
The regulation of GSH in ferroptosis GSH is a key antioxidant against ferroptosis
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