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As mentioned above, hyperglycemia induces oxidative stress, which may lead to endothelial dysfunction, mainly manifested in: Endothelial nitric oxide synthase (eNOS) activity is inhibited, NO synthesis is reduced, thrombosis factors (such as tissue factor and plasminogen activator inhibitors1) or ET-1 production is increased, which induces thrombosis and vasoconstriction, and nuclear factor kappaB (NF-B) and activator protein1 are increased, leading to tissue inflammation (Defeudis et al

Bayberries color index was determined as CIRG = (180 h)(C + L

The RNAi mechanism involves small interfering RNA molecules that efficiently down-regulate desired genes

GABA(B)R expressed on vagal afferent neurones inhibit gastric mechanosensitivity in ferret proximal stomach