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These results demonstrated that injection of the BI1 plasmid effectively increases BI1 protein levels while suppressing Bax expression in ALS mice, resulting in significant inhibition of apoptotic activation (Fig

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The G-type At4g21390 and L-type At4g28350 ( LecRK-VII.2 ) were the most highly induced LecRKs at 1 h and 9 h

This discordant signal may be related to the fact that kidney transplant recipients often have concomitant hypertension and activation of the renin-angiotensin system that may have already contributed to significant adverse cardiac remodeling prior to kidney transplantmaking it less likely to observe differences following kidney transplantation (75, 78)