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The genetic inactivation of the regulatory component in the rate-limiting step of GSH biosynthesis ( GCLM ) or the treatment with L-BSO further depleted endogenous GSH in both ADH5 and WT cells, denoting that the mechanism by which ADH5 contributes to GSH homeostasis is downstream GSH synthesis

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J Neurochem 73:889902, PMID: 10461878 Varga V, Janky R, Marnela KM, Gulys J, Kontro P, Oja SS (1989) Displacement of excitatory amino acid receptor ligands by acidic oligopeptides

Functional interaction between morphine and central amygdala cannabinoid CB1 receptors in the acquisition and expression of conditioned place preference