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Overall, the review highlights metabolism as a key determinant of GBM pathophysiology and a promising target for therapeutic innovation, emphasizing the importance of continued translational research to identify and exploit context-specific metabolic vulnerabilities in this highly lethal disease

Right panel : The corresponding site in hSERT

Colonic Organoids Display Genotype-Specific Sensitivity to Molidustat It has previously been reported that Molidustat treatment reduces tumour growth in an in vivo xenograft model of breast cancer 17

Liu J, Lin S, Dang J, et al
