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This persistent pain is attributed to a combination of inflammatory and non-inflammatory mechanisms, with peripheral and central sensitization being implicated as key contributors (Biddle and Sofat 2020)

However, all of the abovementioned changes occurred independently of any change in body composition or hemodynamic parameters

An overview

In leptin-deficient obese mice, CB1-expressing presynaptic neurons change from glutamatergic to predominantly GABAergic in the lateral hypothalamic area, and because CB1 is associated with G i/o protein, this remodeling results in increased orexin-A, an orexigenic peptide (Cristino et al., 2013)