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Unlike symptomatic treatments (gabapentinoids, duloxetine), ALC promotes actual nerve fiber regeneration and addresses underlying pathophysiology.[17][2] This disease-modifying potential makes it particularly valuable for early intervention

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170 In periosteum-derived skeletal progenitors, pharmacological inhibition of Complex III of the electron transport chain increased succinate and 2-hydroxyglutarate levels that inhibited ten-eleven translocation (TET) DNA demethylase activity, causing wide-spread DNA hypermethylation in the genome