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Interleukin-1 antagonism in type 1 diabetes of recent onset: two multicentre, randomised, double-blind, placebo-controlled trials

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Diabetic ED develops as a consequence of a combination of endothelial dysfunction, autonomic neuropathy, and vascular complications 6,7 , and we have previously demonstrated that PBM can contribute to the regeneration of neurons in a model of cavernous injury 4 , on the basis of which we accordingly postulated that PBM would might also have similar effects on neuronal cells and peripheral nerves in the penile tissues of mice with STZ-induced diabetes