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( 2 S concentration in penile tissue is related to the occurrence of BCNI-ED, and exogenous H 2 S inhibits the phenotypic transformation of CCSMCs and improves the erectile function of BCNI rats by inhibiting the RhoA/ROCK1 pathway and consequently affecting its downstream factors CDK2, Cyclin E1 and P27 kip1

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Tumor-specific targeting by IB-DNQ and Rucaparib combination treatment provides strong support for their potential use as a clinical therapy for patients with TNBC and our findings provide a compendium of cellular signaling changes with short single-agent and combination treatments defining the pathways that are highly affected that could contribute to cancer resistance mechanisms

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