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Furthermore, in the Diabetes Care study, curcumin notably appeared to help improve the function of beta cells, which research shows help produce the hormone insulin

The therapeutic potential for GGC to increase GSH is related to the second GSH biosynthetic enzyme, glutathione synthetase (GS) which is not regulated by non-allosteric feedback inhibition by GSH

Notably, ferroptotic cell death, regardless the mechanisms of activation, can be blocked by iron chelators, indicating a central role of iron in the regulation and execution of ferroptosis

This effect is particularly relevant in the context of chronic inflammation, where barrier dysfunction contributes to disease progression