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Additionally, the mTOR, IL-17, NOD-like receptor and HIF-1 signaling pathways were involved (Supplementary Fig
Moreover, a prolonged inhibition was detected in the excitation of spinal neurons generated by SP (Fig
Immunosenescence is typically linked to autoimmune diseases and inflammation, and it can significantly speed up the aging process
125 Furthermore, IDH1 silencing results in decreased NADPH and -KG levels, with the increased ROS levels, leading to cancer cell apoptosis in NSCLC
