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Description
It is commonly studied in wound healing, tendon repair, gut protection, and inflammation research models
Importantly, mutant SOD1 has been demonstrated to decrease the expression of antioxidant transcription factor NRF2 and its dependent target genes in an established NSC134 cellular model for SOD1-associated ALS (Kirby et al., 2005)
Additional funding was provided by the National Science and Technology Major Project [no
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