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Chen XD, Zhao J, Yang X, Zhou BW, Yan Z, Liu WF

Exposure to these infectious pathogens and inflammatory stimuli can profoundly affect the brain and behavior ( Toxoplasma gondii infection, which, in animal models, alters behavior and increases the release of neurotransmitters such as dopamine, and the amount of dopamine release is correlated with the number of infected cells ( + T cells, which are crucial for sustaining lasting immunity, have been found to be downregulated in schizophrenia patients ( Toxoplasma gondii infection has been shown to affect the gut microbiome in mice ( Table 1 Clinical evidence for the association between infectious pathogens and schizophrenia

In line with that, Superoxide dismutase 1 ( Sod1) knockout mice and mice expressing mutated Sod1 exhibit increased PC in sciatic nerve and skeletal muscle, respectively ( Gpx4 haploinsufficient mice fed a high fat-high sucrose diet exhibit increased 4-HNE adducts in liver and heart ( The second line of defense involves direct detoxification of free RLAs by specialized phase I and II metabolic enzymes ( +/ NAD(P)H cofactors to oxidize RLAs to their corresponding acids or reduce them to alcohol

Although it was not able to clearly elucidate the mechanism underlying the decrease in autolysosomes relative to autophagosomes in this moment, it can be hypothesized that the fusion of autophagosomes with lysosomes may be disrupted due to lysosome dysregulation