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Description
Molecular evidences for a role of HSV-1 in multiple sclerosis clinical acute attack

PT cells lacking show decreased basal OCR and slightly increased extracellular acidification rate (ECAR) in line with decreased mitochondrial coupling efficiency, suggesting that TGF- signaling inhibition directly impacts the PT cells capacity to efficiently produce ATP via OXPHOS

IGF-binding proteins are multifunctional and act via IGF-dependent and -independent mechanisms
Conventional redox-sensitive GFPs (roGFPs) are limited by undefined specificity and slow response to changes in redox potential
