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Phase-I metabolites are highly apt to form adducts with proteins and nucleic acids because of their electrophilic character

Moreover, loss of function of PINK1, disrupts mitochondrial autophagy and contributes to an increase in ROS levels (Requejo-Aguilar et al., 2014)

The druglike and orally bioavailable compounds identified herein are active in animal models of schistosomiasis with a broader range of developmental stages targeted than the drug of choice, PZQ

A provider should review these alongside oxidative stress markers to determine whether antioxidant support is clinically justified for your specific metabolic profile